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Bittner et al. also observed that high Lp(a) identified post-ACS patients who were at higher risk, particularly for Lp(a) ≥60 mg/dl. The number needed to treat with alirocumab was much lower for subjects in the top Lp(a) quartile versus the bottom Lp(a) quartile (number needed to treat: 49 vs. 238), which was consistent with the results for.
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After accounting for LDL-C, each type of Lp(a) test provides important incremental information regarding the risk for MACE in patients with recent ACS and risk reduction with alirocumab. In terms of choosing a commercially available Lp(a) immunoassay for individual patient prognosis, both can provide comparable clinical utility for risk assessment.
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This was particularly meaningful for patients with high baseline Lp(a) levels. This may represent a novel therapeutic target among ACS patients. Interestingly, elevated baseline hsCRP levels amplified the relationship of Lp(a) to adverse cardiovascular outcomes. Alirocumab had the highest benefit among patients with elevated hsCRP and Lp(a).
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The precise role of Lipoprotein associated phospholipase A2 (Lp-PlA2) in the pathogenesis of acute coronary syndromes (ACS) and in the prediction of future cardiovascular events is still debated. So far, few data exist on the variations of Lp-PlA2 activity in ACS and especially in NSTE-ACS vs. STEMI patients, where thrombotic and atherosclerotic mechanisms could play a differential role. The.
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In patients presenting with ACS,. (Lp(a)) and LDL-C on major adverse cardiovascular events. 69 The authors found that both baseline levels and relative reductions of Lp(a) and LDL-C were associated independently with a reduction in major adverse cardiovascular events. While providing mechanistic insight into the effect of alirocumab, these.
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Lipoprotein (a) Proprotein Convertase 9. Antibodies, Monoclonal, Humanized. Lipoprotein (a) Proprotein Convertase 9. K24 HL136852/HL/NHLBI NIH HHS/United States. R01 HL134811/HL/NHLBI NIH HHS/United States. Lp (a)'s Odyssey: Should We Measure Lp (a) Post-ACS and What Should We Do With the Results?
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Objective: The current study was to evaluate the association of Lipoprotein (a) [Lp(a)] and in-hospital outcomes in patients with acute coronary syndrome (ACS) undergoing percutaneous coronary intervention (PCI).Methods: ACS patients undergoing PCI were retrospectively enrolled.Based on Lp(a) level, patients were divided into low (<30 mg/dL) and high (≥30 mg/dL) Lp(a) groups.
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Background and aims: High levels of lipoprotein(a) [Lp(a)] are associated with increased risk of acute coronary syndrome (ACS). We explored whether Lp(a) exhibits a stronger association with premature ACS. Methods: A case-control study was conducted; 1457 patients with a history of ACS (54.8 ± 13 years, 86% males) and 2090 age-sex matched adults free of cardiovascular disease were enrolled.
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Lp(a) level measured at least once in all adults and in youth with a history of ischemic stroke or a family history of premature ASCVD or elevated Lp(a) level and no other known risk factors. 2021 CCS dyslipidemia guideline. Measure once in a person's lifetime as part of initial lipid screening. 2019 NLA scientific statement on Lp(a).
(DOC) Lp Acs Lola Ci Akademik DOKUMEN.TIPS
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Lipoprotein (a) (Lp [a]) is a promising biomarker to help refine current strategies of ASCVD risk assessment, and it is estimated to be elevated in approximately 20% of the world's population. Research on Lp (a) suggests it has added value in preventive medicine, and it is time for cardiologists and cardiovascular team members to consider using.
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Low-density lipoprotein cholesterol (LDL-C) is a traditional and important risk factor for atherosclerotic cardiovascular disease (CVD). Recently, lipoprotein (a) (lp(a)) attracts considerable attention as a residual risk factor for CVD. However, the roles of lp(a) in acute coronary syndrome (ACS) patients with well-controlled LDL-C (≤1.8mmol/L) after percutaneous coronary intervention (PCI.
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Elevated Lipoprotein (a) ( Lp (a)) is a causal, independent, genetic risk factor for CVD (cardiovascular disease) and heart attack. Elevated Lp (a) plays a crucial role in the growth and destabilization of plaques resulting in ACS. 1-5. Large quantities of Lp (a) can be detected in culprit lesions from patients with ACS compared to patients.
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Lp(a) was measured after ten hours fasting. Lp(a) >30 nmol/l) were considered as high. Data were entered and analyzed in SPSS 17. Independent sample t-test was used to compare the mean lipoprotein (a) in cases and controls.. ≤30mg/dl, high risk for ACS: 31-50 mg/dl, very high risk for ACS: >50 mg/dl. Cardiovascular risk is increased by 1.6.
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Lipoprotein-associated phospholipase A2 (Lp-PLA2) is considered to be a risk factor for acute coronary syndrome (ACS), but this remains controversial. This study investigated the role of Lp-PLA2.
